Dyslexia – despite conventional wisdom – is actually a genetic problem. Our genetic screening solution can identify children at high risk before they enter school and struggle with reading curriculum.  Our test is highly scalable, non-invasive (saliva sample), and very cost-effective.

Multiple independent twin and family studies show that the heritability of dyslexia is between 50% and 80% - that is to say that 50% to 80% of the variance in reading performance can be explained by genetic factors.

12+ years of academic research covering the genetics of Learning Disabilities has been funded and supported by three NIH grants, one NIH Small Business Innovation Research grant and a private foundation.

In early 2000s, a number of independent labs identified the reading disability (dyslexia) gene locus on chromosome 6 (DYX2). The originating lab was the first to generate high-resolution genetic markers, genetic association maps, and gene expression maps of DYX2. These studies led to the identification of DCDC2, a dyslexia gene that was cited by the journal Science as the 5th top breakthrough of 2005. The lab performed an NIH funded clinical study of DCDC2 and other genes related to reading and language using a database of 10,000+ children and mothers. These studies identified the transcriptional control element called READ1, and READ1 alleles that are detrimental and protective for reading disability and language impairment. 

Research and testing on a wholly-separate 6000+ patient samples has validated the utility of our markers in predicting Learning Disabilities.  The findings have proven to be reliable and consistent.